Respiratory applications of telemedicine.

نویسنده

  • Christopher B Cooper
چکیده

oxidative metabolism and interleukin-8 production in human neutrophils exposed to azithromycin: effects of Streptococcus pneumoniae. J Antimicrob Chemother 2000;46:19–26. 30. Yamasawa H, Oshikawa K, Ohno S, et al. Macrolides inhibit epithelial cell-mediated neutrophil survival by modulating granulocyte macrophage colonystimulating factor release. Am J Respir Cell Mol Biol 2004;30:569–75. 31. Hodge S, Hodge G, Brozyna S, et al. Azithromycin increases phagocytosis of apoptotic bronchial epithelial cells by alveolar macrophages. Eur Respir J 2006;28:486–95. 32. Hodge S, Hodge G, Jersmann H, et al. Azithromycin improves macrophage phagocytic function and expression of mannose receptor in chronic obstructive pulmonary disease. Am J Respir Crit Care Med 2008;178:139–48. 33. Seemungal TAR, Wilkinson TMA, Perera W, et al. Long term macrolide therapy decreases exacerbations of COPD. Am J Respir Crit Care Med 2007;175:A764. 34. Abdelghaffar H, Vazifeh D, Labro MT. Erythromycin Aderived macrolides modify the functional activities of human neutrophils by altering the phospholipase Dphosphatidate phosphohydrolase transduction pathway: L-cladinose is involved both in alterations of neutrophil functions and modulation of this transductional pathway. J Immunol 1997;159:3995–4005.

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عنوان ژورنال:
  • Thorax

دوره 64 3  شماره 

صفحات  -

تاریخ انتشار 2009